THE GOOD CARBS COOKBOOK
The Good Carbs Cookbook (by Alan Barclay, Kate McGhie and Philippa Sandall) published by Murdoch Books
helps you choose the best fruits, vegetables, beans, peas, lentils,
seeds, nuts and grains and explains how to use them in 100 refreshingly
nourishing recipes to enjoy every day, for breakfast, brunch, lunch,
dinner and dessert. The recipes are easy to prepare, (mostly) quick to
cook, long in flavour and full of sustaining goodness, so you feel
fuller for longer. There is a nutritional analysis for each recipe and
tips and helpful hints for the novice, nervous, curious or time-starved
cook.
OMELETTE WITH GARDEN PEAS, FETA AND MINT
There
are times when the yearning for an omelette is overwhelming, but for
some reason, many cooks shy away from this gloriously simple light meal.
Practice makes perfect, but don’t be too fussed about how it looks when
turned out. The refreshing, lively filling makes up for any
imperfection in the looks department. Ring the filling changes as you
fancy: cooked prawns, flaked cooked fish, diced fresh tomato and plenty
of fresh chopped herbs are all delicious options. And if you are a
family of smaller eaters, it will easily serve three people as a light
meal with a slice of grainy bread and a garden salad with ripe-red
tomatoes. Preparation time: 15 minutes • Cooking time: 8 minutes •
Serves: 2
⅔ cup (100g/3½oz) cooked peas
1 tablespoon olive oil
1 teaspoon finely grated lemon zest
2 spring onions (scallions), finely sliced
6 eggs (60g/2oz eggs)
Salt flakes and freshly ground pepper
2 tablespoons butter
½ cup (60g/2oz) crumbled feta
3 tablespoons shredded mint leaves
Put
the peas in a bowl with the oil, lemon zest, and spring onions. • Whisk
the eggs with salt and pepper in a bowl. Heat the butter in a large
non-stick pan over medium heat. When the butter foams and turns nut
brown, add eggs and gently cook, without stirring for about 3 minutes or
until large curds form. Reduce the heat to medium-low and using a broad
spatula gently push the eggs around in the pan until they are almost
set. Spoon the pea mixture, feta and mint over the top, and with the aid
of the spatula, gently roll the omelette up and over the filling. This
is easier if you hold the pan at a slight angle to assist the rolling.
Cook for 1 minute more and then slide the omelette onto a plate. Divide
into two and serve topped with extra mint and spring onion if you wish.
Per serve
2350kJ/560
calories; 28g protein; 47g fat (includes 21g saturated fat; saturated :
unsaturated fat ratio 0.81); 6g available carbs (includes 2g sugars and
4g starches); 4g fibre; 740mg sodium; 310mg potassium; sodium :
potassium ratio 2.4
1 June 2018
IN THE GI NEWS KITCHEN
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COPYRIGHT AND PERMISSION
This website and all information, data, documents,
pages and images it contains is copyright under the Copyright Act 1968
(Commonwealth of Australia) (as amended) and the copyright laws of all
member countries of the Berne Union and the Universal Copyright
Convention.Copyright in the website and in material prepared by GI News
is owned by Glycemic Index Foundation. Copyright in quotations, images
from published works and photo libraries, and materials contributed by
third parties including our regular contributors Alan Barclay, Jennie
Brand-Miller, Nick Fuller and Nicole Senior is owned by the respective
authors or agencies, as credited.
GI News encourages
the availability, dissemination and exchange of public information. You
may include a link to GI News on your website. You may also copy,
distribute, display, download and otherwise freely deal only with
material owned by GI News, on the condition that you include the
copyright notice “© GI News, University of Sydney” on all uses and
prominently credit the source as being GI News and include a link back
to ginews.blogspot.com.au. You must, however, obtain permission from GI News if you wish to do the following:
- charge others for access to the work
- include all or part of the work in advertising or a product for sale, or
- modify the work.
This permission does not extend to material contributed and owned by other parties. We strongly recommend that you refer to the copyright statements at their respective websites and seek their permission before making use of any such material, whether images or text. Please contact GI News if you are in doubt as to the ownership of any material.
Nutritional analysis To analyse Australian foods, beverages, processed products and recipes, we use FoodWorks which contains the AusNut and Nuttab databases. If necessary, this is supplemented with data from www.calorieking.com.au or http://ndb.nal.usda.gov/ndb/search.
Disclaimer GI News endeavours to check the veracity of news stories cited in this free e-newsletter by referring to the primary source, but cannot be held responsible for inaccuracies in the articles so published. GI News provides links to other World Wide Web sites as a convenience to users, but cannot be held responsible for the content or availability of these sites. All recipes that are included within GI News have been analysed however they have not been tested for their glycemic index properties by an accredited laboratory according to the ISO standards.
© ®™ The University of Sydney, Australia
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1 May 2018
GI News - May 2018
GI News is published by the University of Sydney,
School of Life and Environmental Sciences and the Charles Perkins
Centre. Our goal is to help people choose the high-quality carbs that
are digested at a rate that our bodies can comfortably accommodate and
to share the latest scientific findings on food and diet with a
particular focus on carbohydrates, dietary fibres, blood glucose and the
glycemic index.
Publisher: Professor Jennie Brand-Miller, AM, PhD, FAIFST, FNSA
Editor: Philippa Sandall
Scientific Editor/Managing Editor: Alan Barclay, PhD, APD AN
Contact GI News: glycemic.index@gmail.com
Sydney University Glycemic Index Research Service
Manager: Fiona Atkinson, PhD, APD AN
Contact: sugirs.manager@sydney.edu.au
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FOOD FOR THOUGHT
WHY IS PUSS PORTLY?
As our waistlines have expanded, so
have those of our pets. The Association for Pet Obesity Prevention’s
ninth annual clinical survey (2016) reports that nearly 54 percent of
dogs and 59 percent of cats were clinically overweight or obese in the
US. To put some numbers on that, they reckon that equals an estimated
41.9 million dogs and 50.5 million cats (based on 2016 pet population
projections provided by the American Pet Products Association). Being
overweight puts puss and puppy at an increased risk for weight-related
disorders such as type 2 diabetes, osteoarthritis, hypertension and many
cancers.
A
recent Swedish cross-sectional study using data from medical records
for cats visiting an academic medical centre and from a questionnaire on
insured cats found that the factors associated with increased risk of
puss being obese were: “Eating predominantly dry food, being a greedy
eater, and inactivity”.
What’s the ideal weight for a
cat? The Cat Bible author and Radio Pet Lady, Tracie Hotchner, says “it
is hard to judge since cats come in so many shapes and sizes. However,
if your cat has a belly that hangs down and swings when she walks, you
need to make some plans to reduce her weight. Oftentimes this will
simply mean removing all dry food and feeding canned or raw instead,
which research shows is the best diet for every cat.”
Dry
food is not appropriate for domestic cats she says. “Numerous
veterinarians who share my dismay over the widespread use of dry food
are concerned about a cat's digestive system being challenged to process
foods it is not designed to eat. Cats are not ‘little dogs’ yet a dry
food developed for canines was then manipulated to give to cats.”
Hotchner views dry food for cats as an addictive harmful source of
nutrition which she calls ‘kitty crack’ as she believes it encourages
felines to consume carbohydrate-heavy plant-based food sources which
their body is not designed to digest and metabolize. Keep in mind the
wild ancestors of puss snoozing on the sofa were obligate carnivores and
their diet was essentially the small animals they hunted. Despite
appearances, the domestic cat still closely resembles its wild ancestor.
Who
thought of dried food for cats and dogs? And when? It has a fascinating
past as GI News editor Philippa Sandall discovered researching
Seafurrers, her book on ships’ cats. The story goes that in the late
1850s, an Ohio electrician named James Spratt journeyed to London to
sell lightning rods. He noticed dogs hanging around the docks at
Portsmouth tucking into scraps of hardtack (ship’s biscuit) and had a
eureka moment. He patented a similar biscuit for dogs (they can digest
carbohydrate-based foods) and the rest is history. Spratt’s Patent Meal
Fibrine Dog Cakes were a baked mixture of wheat, beet root, and
vegetables bound together with beef blood. Dried food (kibble) for cats
followed.
To whet your appetite, here’s a World War 2
“dry food incident” reported by a 17-year-old Massachusetts seaman who
saved the ship’s cat after they were torpedoed. “We were in the lifeboat
seven and a half days with not much to eat besides hardtack,” he said.
“The cat didn’t like hardtack and wouldn’t eat a bite until some flying
fish landed in the boat. Before we got to shore, though, she ate
hardtack and liked it.” It’s likely the lifeboat lad improvised a grainy
seafood salad to tempt puss’s taste buds (and his own) by tossing
crumbled hardtack with flying fish flakes moistened with a little water
and puss focussed her attention on the fishy bits surmises Seafurrers
author Philippa Sandall.
Read more:
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WHAT’S NEW?
DIABETES IN CATS
Diabetes in cats resembles type 2
diabetes in people. The causes aren’t fully understood, but both genetic
and environmental factors are believed to contribute. However, for
those quick to point the wagging finger at “sugar” causing obesity and
diabetes, cats don’t eat sugar. They don’t have a sweet tooth. A small
Swedish case-control study using a web‐based questionnaire sent to
owners of cats with diabetes and cats without diabetes (the control)
found indoor confinement, being a greedy eater, and being overweight
were associated with an increased risk of diabetes. As dry food is
commonly fed to cats worldwide, “the association found between dry food
and an increased risk of diabetes in cats assessed as normal weight by
owners warrants further attention” say the authors.
In
Perspectives this month, Alan Barclay looks at the pancreas, what it is
and what it does, and why understanding the causes of diabetes are
complicated in people let alone in cats.
Read more:
A high fibre diet rich in good carbs (fruit, veg, beans and grains) can help people with type 2 diabetes manage their blood glucose levels – and it seems to do this by changing the bacteria in the gut. The findings of a recent study showed a diversified high-fibre diet can promote some 15 strains of gut bacteria that produce short-chain fatty acids that can help in reducing inflammation in the gut, help regulate hunger and also provide energy to gut cells. “It’s early days,” says study leader Prof Liping Zhao from Rutgers University, “but it lays the foundation and opens the possibility that fibres targeting this group of gut bacteria could eventually become a major part of your diet and your treatment”. The research reinforces the fact that eating certain kinds of carbohydrate foods rich in dietary fibres can help restore the gut microbiota responsible for better digestion and overall health.
Read more:
IT’S NOT JUST IN YOUR HEAD: SEROTONIN AND OBESITY
Maybe it’s not on the tip of every tongue writes Ted Kyle in ConscienHealth. But serotonin is a bit more familiar than most neurotransmitters. Most people think of it as a happy hormone for the central nervous system that becomes depleted in a state of depression. However, the gastrointestinal system has far more of it than the central nervous system. And new research now tells us that in the small intestine, this substance might influence obesity and metabolic health.
Scientists have long known that serotonin in the brain plays a role in eating behaviour. Food intake is higher when levels of this hormone are lower in the brain. But animal studies have suggested a very different relationship between serotonin and obesity in the gut. There, it seems to promote obesity and higher blood glucose levels. Now, we have confirmation in humans that this is true. Richard Young and colleagues showed that the small intestines of people with obesity produce more serotonin. In fact, the levels were twice as high when compared to normal controls. The gut secretes this hormone in response to glucose and it appears to play a role in developing obesity and diabetes.
This research is important for two related reasons says Kyle. First, it gives us more insight into how both obesity and diabetes develop, and why some people are more susceptible than others. In their research, Young et al found more cells that produce serotonin in the small intestines of people with obesity than in those at a normal weight. With a better understanding of this pathway, we might have a promising new target for treating obesity and diabetes. Says Young: “This has revealed new ways that we may be able to control the release of serotonin from within the gut, and in turn, further improve the outlook for people living with obesity.”
Read more:
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PERSPECTIVES WITH DR ALAN BARCLAY
THE PANCREAS – UNLOVED, BUT ESSENTIAL
The pancreas is an
essential organ responsible for both the digestion of food and blood
glucose regulation. It was first identified by Greek anatomist and
surgeon, Herophilus, around 2300 years ago. A few hundred years later,
Rufus of Ephesus, another Greek anatomist, gave the pancreas its name.
“Pancreas” originally meant sweetbread, a name that is still commonly
used in culinary circles for calf or lamb pancreas.
The
pancreas is located behind the stomach in the upper left part of the
abdomen. It is surrounded by other organs including the stomach, small
intestine (duodenum), liver, and spleen. It is spongy, about 15–25cm
(6–10in) long, 2.5cm (1in) thick, and is shaped a bit like a flattened
pear or a fish extended horizontally across the abdomen. The bulk (95%)
of the pancreas consists of tissues and cells that produce pancreatic
secretions for the digestion of carbohydrates, fats and proteins. The
remainder consists of little islands of cells called islets of
Langerhans. These look a bit like small bunches of grapes and produce
hormones that regulate blood glucose and help regulate pancreatic
digestive secretions.
Source: www.medicalook.com
Food digestion
Once food has been chewed in the mouth, then mulched and partially
digested in the stomach by acids, it is released into the first part of
the small intestine known as the duodenum. The pancreas then releases
its own digestive juices and enzymes into the partially digested food,
via a small duct connected to the duodenum. Pancreatic juices contain
enzymes that help breakdown carbohydrate, fat and protein. They are
activated once they reach the duodenum to prevent the protein-digesting
enzyme trypsin from breaking down the proteins in the pancreas itself,
or in its duct. Other enzymes produced by the pancreas and released into
the duodenum include amylase (to break down starches and maltodextrins
into sugars) and lipase (to break down fats into monoglycerols and fatty
acids). The pancreas also secretes sodium bicarbonate, which helps to
neutralise the stomach acids in the partially digested food.
Blood glucose hormones
Two of the most important pancreatic hormones are insulin produced by
beta cells and glucagon produced by alpha cells in the islets of
Langerhans which manufacture and release these hormones directly into
the bloodstream.
Insulin regulates the metabolism of
carbohydrates, fats and protein by promoting the absorption of glucose
from the blood into liver, fat and muscle cells. In these cells the
absorbed glucose is converted into either glycogen (a kind of starch
found in the liver and muscles) via a process known as glycogenesis or
fats (triglycerides) via lipogenesis. Circulating insulin also affects
the synthesis of proteins in a wide variety of cells and tissues. It is
therefore an anabolic hormone, promoting the conversion of small
molecules in the blood into large molecules inside the cells.
Glucagon
stimulates the liver to break down glycogen into glucose, which is then
released into the blood. It also activates gluconeogenesis, the
conversion of certain amino acids from proteins into glucose. Finally,
it facilitates the breakdown of stored fat (triglycerides) into fatty
acids for use as fuel by cells. It is therefore a catabolic hormone,
promoting the breakdown of large molecules in cells into smaller
molecules in the blood.
Pancreatic beta cells are
sensitive to blood glucose concentrations. When glucose levels are high,
they secrete insulin into the bloodstream and when glucose levels are
low, secretion of insulin is inhibited. On the other hand, alpha cells
secrete glucagon into the blood in the opposite manner to insulin: when
blood glucose levels are low, or in response to vigorous exercise,
secretion is increased, and when blood glucose levels are high,
secretion is decreased.
The secretion of insulin and
glucagon into the blood in response to changes in blood glucose
concentrations is the primary mechanism of blood glucose homeostasis. In
other words, the two hormones work in partnership with each other to
keep blood glucose levels balanced. Optimal maintenance of blood glucose
levels is critical to the functioning of key organs including the brain
and nervous system, liver, and kidneys.
If the beta
cells are destroyed by an autoimmune reaction, insulin can no longer be
synthesized or secreted into the blood in sufficient quantities. This
results in the development of type 1 diabetes. In type 2 diabetes, the
destruction of beta cells is less pronounced than in type 1 diabetes and
is not primarily due to an autoimmune process. The exact cause of type 2
diabetes is not fully understood but people have a reduced number of
islet beta cells, and of those that survive there is a reduced secretory
function, and there is also frequently (but not always) peripheral
tissue insulin resistance (the insulin that is produced does not work as
efficiently in the target cells as it should). Type 2 diabetes is also
characterized by high rates of glucagon secretion which are less
responsive to the concentration of glucose in the blood, but insulin is
still secreted into the blood in response to concomitantly increasing
blood glucose concentrations. As a result, insulin levels are typically
much higher than they are in people without type 2 diabetes.
Read more:
Alan Barclay, PhD is a consultant dietitian. He worked for Diabetes Australia (NSW) from 1998–2014 . He is author/co-author of more than 30 scientific publications, and author/co-author of The good Carbs Cookbook (Murdoch Books), Reversing Diabetes (Murdoch Books), The Low GI Diet: Managing Type 2 Diabetes (Hachette Australia) and The Ultimate Guide to Sugars and Sweeteners (The Experiment, New York).
Contact: You can follow him on Twitter or check out his website.
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KEEPING IT GREEN – EATING FOR BODY AND PLANET
LOW ENERGY LIVING
We use a lot of energy in our everyday
lives. Cast your mind back to the last power outage. Did your heart sink
as you realised that your mobile battery was at 2%, you couldn’t watch
TV or make microwave popcorn and the ice-cream in the fridge melted? The
minor and temporary inconvenience of a power outage is the tip of a
very large energy iceberg.
Each year we are using more
and more energy. One way of measuring how much more we are using is
Overshoot Day. In 2017, August 2 was Overshoot Day says the Global Footprint Network.
What this means is that in just over seven months, we, the world’s
population, had used up the natural resources such as food and fuel
Earth can generate in twelve months. As Overshoot Day moves earlier and
earlier each year, we dip more and more into the super fund of natural
resources we should be saving for our children’s and their children’s
future. What can we do to make a difference? We can start with adopting
some everyday habits that help to reduce our energy use. We don’t
necessarily have to go without; but we need to be more aware of the
energy we use in our daily lives, be more energy efficient and waste
less.
Our energy-saving tips
Transport
- Walk, ride a bike, carpool or take public transport – it saves money and reduces your carbon footprint as well as increasing your activity levels. Talk about win, win, win!
- Buy healthy whole foods such as oats, brown rice, vegetables, fruit, milk, fresh meat, legumes and eggs, and cut back on (cut out?) highly processed packaged foods that require more energy to produce in the first place and are likely high in salt, saturated fats and highly refined carbohydrates (starches and sugars) that spike BGLs.
- Plan your meals and shopping trips to avoid emergency fast food drive-throughs and pizza deliveries.
- Adjust your air-conditioning thermostat to more moderate settings to make it use less energy, such as 18–20 degrees Celsius in winter and 25–27 degrees in summer.
- Wear more clothes in winter to save on heating (and remember keeping yourself warm uses kilojoules/calories and every little bit helps).
- Close the door on rooms you’re not using and exclude draughts.
- To save energy on water-heating, wash clothes in cold water, only run the dishwasher when its full (and in the middle of the night for off-peak energy pricing), keep showers short and install a water-saving shower head and flow-limiters on taps.
- Air-dry clothes rather than use a clothes dryer.
- Ensure your fridge is set to the correct temperature – around 3–4 degrees Celsius and get rid of that extra fridge – it’s costing you a lot to run.
- Don’t open the fridge door too much so it doesn’t have to work as hard to stay cold.
- Use the BBQ outside on hot days – cooking inside heats the house and makes your cooling system work harder.
- Use your microwave oven or pressure cooker rather than your oven – it is more energy efficient.
- Defrost frozen foods in the fridge overnight instead of in the microwave.
- Use the correct size burner for your saucepan – excess heat wastes energy.
- If using your oven, think about cooking two things at once.
Nicole Senior is an Accredited Nutritionist, author, consultant, cook, food enthusiast and mother who strives to make sense of nutrition science and delights in making healthy food delicious. Contact: You can follow her on Twitter, Facebook, Pinterest, Instagram or check out her website.
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GOOD CARBS FOOD FACTS A TO Z
ORANGES
The
golden glow of oranges in the midst of a grey winter lift spirits. It’s
not just colour. Their sweet acidity makes them a versatile ingredient
in the kitchen (variety doesn’t matter so much, although some recipes
specify blood oranges or Seville oranges for colour or flavour). Whole
oranges (skin and all), segments, juice and zest are used in every
category of cooking from salads, soups, sauces, meat, fish, poultry to
desserts and baking.
As citrus fruit does not ripen
further once it has been picked, choose ripe oranges that smell like
oranges with no hint of fermenting aromas. Heavy fruits with
fine-grained skin are juiciest. Avoid soft, bruised or wrinkled fruit as
well as spongy soft ones. They keep for up to two weeks in the
refrigerator and almost as well at room temperature. They also yield
more juice at room temperature. Their sturdy peel protects them and they
require no further wrapping.
Source: The Good Carbs Cookbook
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IN THE GI NEWS KITCHEN
THE GOOD CARBS COOKBOOK
The Good Carbs Cookbook (by Alan Barclay, Kate McGhie and Philippa Sandall) published by Murdoch Books
helps you choose the best fruits, vegetables, beans, peas, lentils,
seeds, nuts and grains and explains how to use them in 100 refreshingly
nourishing recipes to enjoy every day, for breakfast, brunch, lunch,
dinner and dessert. The recipes are easy to prepare, (mostly) quick to
cook, long in flavour and full of sustaining goodness, so you feel
fuller for longer. There is a nutritional analysis for each recipe and
tips and helpful hints for the novice, nervous, curious or time-starved
cook.
FREGOLA AND BLOOD ORANGE SALAD WITH FENNEL
Every
ingredient in this lively salad says Italy. Red-fleshed blood oranges
were developed in Sicily, while subtle, aniseedy fennel is everywhere in
Italy. Fregola is a type of Sardinian pasta, not dissimilar to
couscous. During processing, the durum wheat flour is mixed with water,
rolled into tiny pebbles, sundried and roasted giving it a rustic
quality. The flavour is improved if the fregola is cooked in a
flavoursome stock. It should take no more than 10–12 minutes of cooking
to be al dente. You can substitute with Israeli (pearl) couscous or orzo
if you wish. Preparation time: 25 minutes •Cooking time: 12 minutes •
Serves: 6
2 cups fregola
4 blood oranges
4 flat anchovy fillets, drained
1 garlic clove, peeled
½ teaspoon chilli flakes
3 tablespoons lemon juice
3 tablespoons olive oil
1 medium fennel bulb, trimmed and thinly sliced
freshly ground pepper
Bring
a medium pan of salted water to the boil and add the fregola. Boil for
about 15 minutes or until cooked through. Drain, refresh under cold
water and set aside to drain and dry. • Slice the peel and white pith
from the oranges and slice into thin rounds. Arrange in a circular
pattern on a serving dish. • Mash the anchovies with the garlic and
chilli flakes and then whisk in the lemon juice and oil. • Put the
well-drained and cooled fregola and the fennel in a bowl, add the
anchovy dressing and toss to combine all the ingredients. Pile the
mixture on top of the oranges and season generously with pepper.
Per serve
1050kJ/250
calories; 6g protein; 10g fat (includes 1.5g saturated fat; saturated :
unsaturated fat ratio 0.18); 33g available carbs (includes 8.5g sugars
and 24.5g starches); 4.5g fibre; 165mg sodium; 315mg potassium; sodium :
potassium ratio 0.52
ANNEKA MANNING’S BAKE CLUB
Anneka Manning is an author, food editor, cooking teacher, home
economist, mother of two and the founder of BakeClub. With over 27
years' experience, she specialises in teaching the ‘why’ behind the
‘how’ of baking, giving home cooks the know-how, understanding and skill
to bake with confidence and success, every time. You can follow her on Twitter, Facebook or check out her website.
MANDARIN ROASTED ALMOND CAKES.
These
gluten-free cakes are a play on the original (and wonderful) Middle
Eastern orange cake from the one-and-only Claudia Roden. The ground
roasted almonds give a lovely ‘toasted’ flavour but you can substitute
pre-ground almond meal. To roast and grind the almonds, spread on an
oven tray and place in an oven preheated to 180°C/350°F for 8-10 minutes
or until aromatic. Cool on the tray before processing in a food
processor until finely ground. Makes: 12 • Prep: 20 minutes (+ 30
minutes simmering and 15 minutes cooling time) • Bake: 15–18 minutes
2 large mandarins
olive oil spray, to grease
½ cup instant polenta
80g (2½oz) natural almonds, roasted, finely ground
½ tsp baking powder
3 eggs, at room temperature
¾ cup raw caster sugar
2 tsp natural vanilla essence or extract
icing sugar, to dust (optional)
Put the mandarins (skin
and all) in a small saucepan and cover with water. Bring to the boil
and simmer for 30 minutes or until very soft when tested with a skewer.
You may need to place a small saucer over the mandarins to keep them
submerged. Remove from the water and set aside to cool slightly. •
Meanwhile preheat the oven to 180°C (350°F). Brush a 12-hole 80ml
(⅓-cup) muffin tin with the melted butter to grease. • Combine the
polenta, roasted almond meal, and baking powder in a medium bowl and mix
well to combine evenly. • Quarter the mandarins and remove and discard
any centre core or seeds. Puree in a food processor or blender until
smooth. • Put the eggs, sugar and vanilla in a medium mixing bowl and
use an electric mixer with a whisk attachment to whisk until very thick
and pale and a ribbon trail forms when the whisk is lifted. Add the
mandarin puree and use a spatula or large metal spoon to fold in until
just combined. Add the polenta mixture and fold together until evenly
combined. • Divide the mixture evenly among the muffin holes (pouring
the mixture from a jug or using a ladle works well). • Bake in preheated
oven for 15-18 minutes or until the cakes are firm to the touch on the
top and cooked when tested with a skewer. Remove from the oven and cool
in the tin for 10 minutes. Use a palette knife to ease the cakes out of
the tin and transfer to a wire rack. Serve warm or at room temperature
dusted with a little icing sugar if you wish.
Per cake
Energy:
560kJ/130cals; Protein 3.5g; Fat 5g (includes 1g saturated fat;
saturated : unsaturated fat ratio 0. 25); Available carbohydrate 18.5g
(includes 15g sugars, 3.5g starches); Fibre 1g; Sodium 40mg; Potassium
95mg; sodium to potassium ratio 0.42
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COPYRIGHT AND PERMISSION
This website and all information, data, documents,
pages and images it contains is copyright under the Copyright Act 1968
(Commonwealth of Australia) (as amended) and the copyright laws of all
member countries of the Berne Union and the Universal Copyright
Convention.Copyright in the website and in material prepared by GI News
is owned by Glycemic Index Foundation. Copyright in quotations, images
from published works and photo libraries, and materials contributed by
third parties including our regular contributors Alan Barclay, Jennie
Brand-Miller, Nick Fuller and Nicole Senior is owned by the respective
authors or agencies, as credited.
GI News encourages
the availability, dissemination and exchange of public information. You
may include a link to GI News on your website. You may also copy,
distribute, display, download and otherwise freely deal only with
material owned by GI News, on the condition that you include the
copyright notice “© GI News, University of Sydney” on all uses and
prominently credit the source as being GI News and include a link back
to ginews.blogspot.com.au. You must, however, obtain permission from GI News if you wish to do the following:
- charge others for access to the work
- include all or part of the work in advertising or a product for sale, or
- modify the work.
This permission does not extend to material contributed and owned by other parties. We strongly recommend that you refer to the copyright statements at their respective websites and seek their permission before making use of any such material, whether images or text. Please contact GI News if you are in doubt as to the ownership of any material.
Nutritional analysis To analyse Australian foods, beverages, processed products and recipes, we use FoodWorks which contains the AusNut and Nuttab databases. If necessary, this is supplemented with data from www.calorieking.com.au or http://ndb.nal.usda.gov/ndb/search.
Disclaimer GI News endeavours to check the veracity of news stories cited in this free e-newsletter by referring to the primary source, but cannot be held responsible for inaccuracies in the articles so published. GI News provides links to other World Wide Web sites as a convenience to users, but cannot be held responsible for the content or availability of these sites. All recipes that are included within GI News have been analysed however they have not been tested for their glycemic index properties by an accredited laboratory according to the ISO standards.
© ®™ The University of Sydney, Australia
Posted by
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at
5:00 am
1 April 2018
GI News - April 2018
GI News is published by the University of Sydney,
School of Life and Environmental Sciences and the Charles Perkins
Centre. Our goal is to help people choose the high-quality carbs that
are digested at a rate that our bodies can comfortably accommodate and
to share the latest scientific findings on food and diet with a
particular focus on carbohydrates, dietary fibres, blood glucose and the
glycemic index.
Publisher: Professor Jennie Brand-Miller, AM, PhD, FAIFST, FNSA
Editor: Philippa Sandall
Scientific Editor/Managing Editor: Alan Barclay, PhD, APD AN
Contact GI News: glycemic.index@gmail.com
Sydney University Glycemic Index Research Service
Manager: Fiona Atkinson, PhD, APD AN
Contact: sugirs.manager@sydney.edu.au
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FOOD FOR THOUGHT
HORSE SENSE FOR WEIGHT LOSS
David
L. Katz, MD, discusses the implications of the JAMA study that compared
low-fat and low-carbohydrate diets for weight loss and health
improvement and also profiled the genes of the participants to determine
if the genetic patterns thought to predict success on a given diet
actually did so. The following edited extract is reproduced with his
permission.
“The study, run by Professor
Christopher Gardner at Stanford University, randomized over 600 adults
to either a fat-restricted or carbohydrate-restricted diet making both
diets as ‘good’ as possible. The result was that significant weight
loss, and health improvement measured in all the usual ways, occurred in
both treatment assignments. Going from the generally poor baseline diet
that prevails in America to either a healthy, low-fat diet that
emphasized vegetables, and whole, minimally processed foods; or to a
healthy low-carbohydrate diet that also emphasized vegetables, and whole
minimally processed foods produced significant, and very comparable
benefits.
Study participants were not told to track or
reduce calories, but by shifting to a much higher-quality diet, they did
so as a by-product. Both groups cut their daily calorie intake by about
500 to 600 kcal daily (2100–2510 kilojoules), and lost the amount of
weight that would portend. They achieved this by eating wholesome foods
in sensible combinations, and by applying some rules and discipline to
diets that had neither at the start.
Genetic markers of
expected success on one type of diet versus the other proved to be of
no value. Weight loss in each diet arm was indistinguishable between
those with a genetic profile saying they should do especially well, and
those with profiles suggesting they should struggle. Nutrigenomically
customized weight loss, despite the buzz it generates, is clearly not
ready for prime time.
The study also found no
relationship between baseline insulin status and success on a given diet
assignment. The high-profile claims that weight loss is all about
reducing carbs to reduce insulin prove to be apocryphal. A shift to
wholesome foods in sensible combinations is effective at lowering weight
and improving health regardless of fat or carbohydrate levels, even
among those with insulin resistance at the start.
Fundamentally,
then, this study suggests that the best way to lose weight and improve
health with diet is not by fixating on macronutrients or calories, but
by eating wholesome foods in some sensible combination, and emphasizing
whole, minimally processed plant foods. The study also indicates that
the fundamentals of generally healthful eating pertain to us all,
regardless of our genes or insulin levels.
These
findings of science may surprise some, but they don’t much surprise me,
and the reason is – sense. Horse sense, in particular. With horses, as
with every species other than our own, we tend to think about the kind
of diet that is generally right for the kind of animal, rather than the
need to customize diet to each individual. Of course, the one does not
preclude the other; horses can all be fed like horses, but some horses
will need extra grain to maintain their weight, some will do better on
certain varieties of hay. But from the start, the focus is on the common
theme of a healthful diet for an entire species, and only after that,
variations on the theme.
The DIETFITS study findings
collectively indicate that the fundamentals of a health-promoting
dietary pattern for Homo sapiens matter more than customizing on the
basis of inter-individual variations. As with horses, the one does not
preclude the other – but the science we own at present better empowers
us to customize diet based on preference, rather than genes. It is good
to know that when it comes to dietary patterns that are best for health,
we do have choices among the variants on a common theme.
Dr.
Katz proudly notes that DIETFITS Principal Investigator, Prof. Gardner,
is a science advisor to his company, DQPN, LLC, devoted to reinventing
dietary intake assessment for the digital age.
About David L. Katz
• Director, Yale University Prevention Research Center; Griffin Hospital
• Immediate Past-President, American College of Lifestyle Medicine
• Senior Medical Advisor, Verywell.com
• Founder, The True Health Initiative
Read more:
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WHAT’S NEW?
VEG OR MED FOR WEIGHTLOSS AND HEART HEALTH?
Take
your pick! According to research published in the American Heart
Association’s journal Circulation, vegetarian and Mediterranean diets
are likely equally effective in reducing the risk of heart disease and
stroke. The study included 107 healthy but overweight participants, ages
18–75, who were randomly assigned to follow either a low-calorie
vegetarian diet (which included dairy and eggs), or a low-calorie
Mediterranean diet for three months. The Mediterranean diet included
poultry, fish and some red meat as well as fruits, vegetables, beans and
whole grains. After three months, the participants switched diets for
another three months. Most participants were able to stay on both diets.
Researchers found participants on either diet:
- lost about 3 pounds (1.4kg) of body fat
- lost about 4 pounds (1.8kg) of weight overall and
- experienced about the same change in body mass index (BMI).
Read more:
VEGAN DIET MAY REDUCE DIABETES RISK
In overweight adults with no history of diabetes, a low-fat, plant-based vegan diet can reduce visceral fat and improve both pancreatic beta-cell function and insulin resistance, potentially decreasing the risk of type 2 diabetes, according to researchers from the Physicians Committee for Responsible Medicine. (Measuring the function of beta cells, which store and release insulin, can help assess future type 2 diabetes risk.)
The study randomly assigned 73 participants with no history of diabetes either to an intervention or control group in a 1:1 ratio. For 16 weeks, participants in the intervention group followed a low-fat vegan diet based on fruits, vegetables, whole grains, and legumes with no calorie limit. The control group made no dietary changes. Neither group changed exercise or medication routines.
Based on mathematical modeling, the researchers determined that those on a plant-based diet increased meal-stimulated insulin secretion and beta-cell glucose sensitivity, compared to those in the control group. The plant-based diet group also experienced a decrease in blood glucose levels both while fasting and during meal tests. Physicians Committee researchers posit that because the intervention group experienced weight loss, including loss of body fat, their fasting insulin resistance decreased (i.e. improved), and their beta-cell function improved as a result.
Read more:
HEALTH BENEFITS OF SWAPPING ANIMAL PROTEINS FOR PLANT PROTEINS
Substituting one to two servings of animal proteins with plant proteins every day could lead to a small reduction in the three main cholesterol markers for cardiovascular disease prevention. The health benefits could be even greater if people combined plant proteins with other cholesterol-lowering foods such as viscous, water soluble fibres from oats, barley and psyllium, and plant sterols, said lead author of the study, Dr John Sievenpiper of St. Michael's Hospital.
The study (a systematic review and meta-analysis of 112 randomized control trials in which people substituted plant proteins for some animal proteins in their diets for at least three weeks) looked at the impact of replacing animal protein with plant protein on three key markers for cholesterol: low-density lipoprotein cholesterol (LDL or “bad” cholesterol, which contributes to fatty build-ups in arteries and raises the risk for heart attack, stroke and peripheral artery disease); non-high density lipoprotein cholesterol (non-HDL-C, or total cholesterol minus HDL or healthy/good cholesterol) and apolipoprotein B (the proteins in bad cholesterol that clog arteries).
Dr. Sievenpiper said the review indicated that replacing one to two servings of animal proteins with plant proteins every day -- primarily soy, nuts and pulses (dried peas and beans, lentils and chickpeas) -- could reduce the main cholesterol markers by about 5 per cent. “That may not sound like much, but because people in North America eat very little plant protein, there is a real opportunity here to make some small changes to our diets and realize the health benefits,” he said.
Read more:
PARENTING IN THE FACE OF OBESITY AND EATING DISORDERS
Childhood obesity is a reality for one in five children in the USA. Five million young people are living with severe obesity. At the same time, half a million US teens are living with an eating disorder. Parents and healthcare providers can’t afford to neglect either of these very real problems.
A study in Pediatrics however reports that encouraging teens to diet can do lasting harm. Teens who receive diet talk from their parents are more likely to have problems with obesity as adults. But that’s not all. These teens are also more likely to have issues with binge eating, unhealthy weight-related behaviours, and body dissatisfaction in adulthood.
Most troubling, though, is the finding that this cycle repeats. As parents, these young adults are more likely to inflict that diet talk on their own children. The cycle repeats. ConscienHealth’s Ted Kyle reports on that confusing four-letter word and what parents should do.
Everyone has a diet, meaning their pattern for eating day after day. But in the context of going on a diet, it becomes a verb – dieting. And that’s where the problem starts. Short-term, highly restrictive diets are not a prescription for long-term health. Nonetheless, people consume great volumes of information about diets: low carb, low fat, Mediterranean, vegetarian, flexitarian, DASH, and the list goes on. Some of these can be the foundation for a sustainable pattern of eating for good health. Others can be severely restrictive and unsustainable. What is clear is that parenting requires attention to the severe harm that four letter word, “diet,” can do. Great harm can result when a parent encourages a child to diet. So, what should parents do and say?
The most basic advice is simple: unconditional love. Beyond that basic impulse for all parenting, the American Academy of Pediatrics (AAP) points to some basics for preventing both obesity and eating disorders in teens.
- Discourage dieting.
- Model a sustainable healthy pattern for eating.
- Promote a positive body image.
- Plan for family meals.
- Talk health, not weight.
- Pay attention to bullying and mistreatment. If you see clues that something is wrong, follow up. Schools and teachers have an obligation to help.
- Seek out real help for obesity. Talk to your pediatrician. Seek out a qualified program with specialists who understand that obesity is a biological issue – not a character flaw or the product of bad parenting.
- Click here for the study by Berge et al
- Click here for a companion commentary
- For guidance from the AAP, click here
- ConscienHealth News
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PERSPECTIVES WITH DR ALAN BARCLAY
DIABETES TIMES FIVE
While we have known about diabetes for
several thousand years, our knowledge and understanding of its causes,
pathophysiology and management is constantly evolving. One of the more
important areas of research that doesn’t receive the attention it
deserves is how we classify the different types. It’s important because
it affects how we manage the condition (e.g., with pharmaceuticals,
dietary strategies, etc) and also how we pay for its management
(Pharmaceutical benefits; Health Insurance rebates, etc).
The
current diabetes classification system, with which most people are
familiar, was developed back in 1979 and lists four types:
- Insulin-dependent or type 1 diabetes
- Non-insulin-dependent or type 2 diabetes
- Gestational diabetes
- Diabetes associated with other syndromes or conditions (e.g., monogenic diabetes syndromes (such as neonatal diabetes and maturity-onset diabetes of the young [MODY]), diseases of the exocrine pancreas (such as cystic fibrosis), and drug- or chemical-induced diabetes (such as in the treatment of HIV/AIDS or after organ transplantation).
Perhaps unsurprisingly, scientists and physicians have been thinking for some time that the current classification system presents challenges to the diagnosis and treatment of people with diabetes, in part due to its conflicting and confounding definitions of type 1, type 2, and LADA.
Back in 2016, a group of US scientists proposed a new B-cell-centric classification of diabetes, based on the presupposition that all diabetes originates from a common denominator – the abnormal pancreatic B-cell. It recognized that interactions between genetically predisposed B-cells with a number of factors, including insulin resistance, susceptibility to environmental influences, and immune dysregulation/inflammation, lead to the range of diabetes sub-types within the spectrum of diabetes. Individually or in concert, and often self-perpetuating, these factors contribute to B-cell stress, dysfunction, or loss through at least 11 distinct pathways. The authors concluded that this classification system enabled “Available, yet underutilized, treatments [to] provide rational choices for more personalized therapies that target the individual mediating pathways of hyperglycemia at work in any given person with diabetes, without the risk of pharmacologically-related hypoglycemia or weight gain or imposing further burden on the B-cells”. To-date, there is little evidence that the B-cell–centric classification of diabetes has been adopted.
The most recent (2018) diabetes classification system has been conceived by Swedish scientists. They developed a 5-cluster system based on the analysis of nearly 9000 people aged 0–96 years who developed diabetes between 2008 and 2016.
Modelling the new system in 500–3500 additional Swedish and Finnish people demonstrated that it was superior to the current diabetes classification system, because it identified people at high risk of diabetic complications (e.g., kidney and eye disease) at diagnosis and provided information about underlying disease mechanisms, thereby guiding choice of therapy.
This new analysis provides another important step towards a more precise, clinically useful stratification of diabetes, representing an important step towards precision medicine in diabetes. It is of course important to note that the new classification system was based on people primarily from northern Europe, with limited non-Scandinavian representation, and the applicability of this strategy to people of other ethnicities needs to be assessed before the model can be adopted globally.
Read more:
- History of diabetes
- The Time Is Right for a New Classification System for Diabetes
- Novel subgroups of adult-onset diabetes and their association with outcomes: a data-driven cluster analysis of six variables
Alan Barclay, PhD is a consultant dietitian. He worked for Diabetes Australia (NSW) from 1998–2014 . He is author/co-author of more than 30 scientific publications, and author/co-author of The good Carbs Cookbook (Murdoch Books), Reversing Diabetes (Murdoch Books), The Low GI Diet: Managing Type 2 Diabetes (Hachette Australia) and The Ultimate Guide to Sugars and Sweeteners (The Experiment, New York).
Contact: You can follow him on Twitter or check out his website.
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KEEPING IT GREEN – EATING FOR BODY AND PLANET
THE LAST STRAW
Single
use straws made sense when contaminated cups were an issue. Thanks to
improved hygiene standards, catching infectious diseases from drinking
vessels is less of an issue but now we have bigger things to worry
about. Disposable plastic drinking straws are an environmental disaster.
The problem with straws Unable to be
recycled, plastic straws are used for 20 minutes at most, then remain
intact for hundreds of years. At the popular Manly Beach in Sydney,
Australia, scuba diver Kasey Turner found 319 straws during a 20-minute
snorkel. Only 1 day later she returned and found an additional 294
straws at the same spot! The following weekend she repeated this
exercise and found a further 150 straws, showing just how quickly these
straws accumulate. And they’re everywhere. According to the Ocean Conservancy’s 2017 Report, straws made the list of top 10 pollutants littering international coastlines.
Besides making a mess, straws do terrible damage to aquatic life. A video posted
in 2015 showing a plastic drinking straw being painfully extracted from
the nostril of a turtle off Costa Rica (the video has a warning that it
may be inappropriate for some users because it is so upsetting). Straws
are even swallowed by seabirds, which then puncture vital organs or
block airways leading to a horrible death.
Compostable
drinking straws are not much better than plastic straws as very few
people compost them, and they are not designed to break down in the
ocean. Even if some plastic straws do manage to break down, they become
smaller microplastics that fish then eat, and we then eat those fish,
plastic and all (yuk). By 2025 it’s been projected there will be more plastic than fish in the oceans.
Straws don’t always suck Straws
do have some great uses. Not only do they give us that Instagram
perfect pout as we sip on a cocktail, they also keep ladies’ lipstick
intact, and make for a less messy drinking experience for kids
(especially in the car). Straws can also reduce contact between sugary
drinks and teeth, which helps prevent cavities and dental erosion. As
straws send liquid to the back of the mouth they reduce flavour exposure
for those suffering from nausea (e.g during chemotherapy or morning
sickness). Bendy straws also make drinking easier for the less-able,
such as the ill, frail, or those with coordination and movement
difficulties.
Do you really need a straw? You
don’t drink beer or wine through a straw, so why not just say “no straw
please”. Your drinks taste just as good (probably better) without a
plastic straw. Perhaps a bit more “slow eating” (and drinking) would
help. Straws tend to go with grab ‘n go drinks with bubbles and sugar.
Let’s face it, we could do with less of these. Taking water with you in a
re-usable bottle has health and environmental benefits.
But I’m a sucker for straws!
For those of you who can’t give up your straws, there are alternatives,
and trendy ones at that. Some bars and cafes already have stainless
steel, re-useable straws. You already use their metal cutlery, so why
not a metal straw? Metal straws have the added benefit of becoming
chilled, which makes your drink even more refreshing, and no plastic
taste. You can even buy a stainless-steel straw for home-use and they
often come with a cleaning brush. Other plastic straw alternatives
include copper, glass and bamboo straws. You can even buy straws from
Harvest Straws made from (would you believe it) straw! That’s right,
straws have come full circle and are now once again made of wheat or rye
straw.
Keeping it green, in a nutshell
- Single-use plastic straws are an environmental disaster and can be devastating to marine life.
- To reduce plastic pollution, asks for drinks with “no straw please”
- For straw devotees, try a reusable drinking straw.
Nicole Senior is an Accredited Nutritionist, author, consultant, cook, food enthusiast and mother who strives to make sense of nutrition science and delights in making healthy food delicious. Contact: You can follow her on Twitter, Facebook, Pinterest, Instagram or check out her website.
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GOOD CARBS FOOD FACTS A TO Z
QUINOA
Quinoa
ticks so many boxes – tasty, quick cooking, packed with nutrition,
gluten free, versatile and colourful it features in soups, salads,
sides, mains, desserts and snacks. It is now grown around the world.
What a tasty success story. While it may currently be the trendiest
grain on the block, it’s not so very long ago that none of us outside
the Altiplano had heard of it. But it has always been the Americas other
major grain. But, unlike corn (maize), the rest of the world forgot
about it for several hundred years. How come the comeback? Three
enthusiastic Americans—Stephen Gorad, Don McKinley and David Cusack of
the Quinoa Corporation (now Ancient Harvest brand) put quinoa on our plates.
The recipe shown in the photograph is Chrissy Freer’s Spice roasted cauliflower, quinoa and petipa pilaf courtesy Australian Healthy Food Guide.
To rinse or not to rinse?
Like
many seeds, quinoa arms itself with bitter-tasting compounds in its
outer skin to deter the unwelcome attention from insects and birds. In
this case it’s saponins. Most quinoa has been treated in some way to
remove the saponins before being packaged for sale, but it’s probably a
good idea to pop the grains in a sieve and run them under cold water
first. Saponins are phytochemicals found in very small amounts in many
plants including veggies (peas, soybeans) and herbs. They tend to pass
straight through us as they are poorly absorbed by our bodies. Heating
destroys them. Naming rights comes from the soapwort plant (Saponaria
officinalis)—its root was used as soap (Latin sapo “soap”) as they have a
natural foaming tendency. With natural cleaning products making a
comeback, there’s growing interest in using saponins for making natural
detergents.
Source: The Good Carbs Cookbook
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IN THE GI NEWS KITCHEN
THE GOOD CARBS COOKBOOK
The Good Carbs Cookbook (by Alan Barclay, Kate McGhie and Philippa Sandall) published by Murdoch Books
helps you choose the best fruits, vegetables, beans, peas, lentils,
seeds, nuts and grains and explains how to use them in 100 refreshingly
nourishing recipes to enjoy every day, for breakfast, brunch, lunch,
dinner and dessert. The recipes are easy to prepare, (mostly) quick to
cook, long in flavour and full of sustaining goodness, so you feel
fuller for longer. There is a nutritional analysis for each recipe and
tips and helpful hints for the novice, nervous, curious or time-starved
cook.
MIXED GRAINS SALAD WITH MARINATED ZUCCHINI
Slow
cooking barley combines with quick-cooking quinoa in this refreshing
salad. The grains soak up the bold flavours giving this Med-style dish
real bite and substance. Serves: 6 • Preparation time: 40 minutes
Cooking time: 30 minutes
6 small zucchini (courgettes)
About ¼ cup olive oil
Salt flakes and freshly ground pepper
2 garlic cloves, crushed
¼ cup red wine vinegar
1 tablespoon caster sugar
3 tablespoons torn mint leaves
3 tablespoons torn parsley leaves
Mixed grains salad
1 cup pearl barley, cooked
1 cup quinoa, rinsed and cooked
1 cup halved red cherry tomatoes
1 cup halved yellow cherry tomatoes
½ cup pitted black olives
½ cup walnut halves
4 large leaves radicchio, roughly torn
2 handfuls torn pale inner leaves of curly endive or rocket
Sea salt flakes and ground pepper to taste (optional)
⅓ cup extra virgin olive oil
1 tablespoon red wine vinegar
To
make the marinated zucchini, wash, trim and cut the zucchini lengthways
into 8 mm (⅜ in) thick slices. Working in batches, heat 2 tablespoons
of the olive oil in a sturdy non-stick pan over medium-high heat.
Arrange the zucchini in a single layer in the pan and cook about 3
minutes each side until well browned. Remove from the pan, arrange in a
shallow dish and sprinkle lightly with salt and pepper. Warm the garlic,
vinegar and sugar in a small pan and pour over the zucchini. Scatter
with mint and parsley, toss lightly and leave at room temperature for 1
hour before serving. • Put the barley, quinoa, tomatoes, olives,
walnuts, radicchio and curly endive into a large bowl with salt and
pepper to taste. Whisk the oil and vinegar together until emulsified,
pour over salad and toss lightly. Serve the salad topped with marinated
zucchini.
Per serve
1590kJ/380 calories;
5g protein; 31g fat (includes 4g saturated fat; saturated : unsaturated
fat ratio 0.15); 18g available carbs (includes 5.5g sugars and 12.5g
starches); 5g fibre; 150mg sodium; 410mg potassium; sodium : potassium
ratio 0.37
REVERSING DIABETES
The latest
research into type 2 diabetes shows that for some people it's possible
to put diabetes into remission and for others they can prevent or at
least delay the complications of diabetes. Reversing Diabetes published by Murdoch Books
explores what these findings mean for you. Drawing on over 20 years of
clinical experience as an Accredited Practising Dietitian, including
nearly 16 years at Diabetes Australia, Dr Alan Barclay combines the
highest quality evidence about the nutritional management and prevention
of diabetes into one easy-to-read book with 70 delicious recipes.
TOMATO, MOZZARELLA AND OLIVE QUINOA PIZZAS
Quinoa
has a light, nutty texture with a slight crunch and will give the pizza
bases a lovely crisp texture. Serves 4 • Preparation 20 minutes + 1
hour resting • Cooking 1 hour
¼ cup quinoa, rinsed
2 teaspoons instant dried yeast
1 cup wholemeal plain flour
½ cup stone-ground plain flour
semolina, for sprinkling
140g (5oz) artichoke hearts in brine, rinsed and halved
100g (3½oz) reduced-fat grated mozzarella cheese
⅓ cup black olives, halved
1 handful basil leaves
200g (7oz) baby English spinach leaves
250g (9oz) baby roma tomatoes, halved
1 small Lebanese (short) cucumber, thinly sliced
2 tablespoons balsamic vinegar
Tomato sauce
2 teaspoons olive oil
1 brown onion, finely chopped
2 garlic cloves, crushed
500g (1lb 2oz) ripe tomatoes, finely chopped
Put
the quinoa and ½ cup water in a saucepan and bring to the boil. Reduce
the heat, cover and simmer for 10–12 minutes or until all the liquid has
evaporated. Transfer to a bowl to cool. • Stir the yeast into 185ml
(6fl oz/¾ cup) tepid water until the yeast has dissolved. Combine the
quinoa and flours in a large bowl. Make a well in the centre, add the
yeast mixture and mix to a soft dough. Turn the dough out onto a lightly
floured surface and knead for 10 minutes or until smooth and elastic.
Return the dough to the lightly oiled bowl, cover with a tea towel (dish
towel) and rest in a warm place for 1 hour or until doubled in size.
To
make the sauce, heat the oil in a saucepan over medium heat. Cook the
onion, stirring, for 4 minutes or until softened. Add the garlic and
stir for 1 minute. Add the tomatoes, reduce the heat to low, cover and
simmer, stirring occasionally, for 15 minutes or until the sauce has
thickened. Remove the lid and cook for 5 minutes or until reduced by
two-thirds. Set aside to cool, then purée using a stick blender.
Preheat
the oven to 220°C (425°F). Sprinkle two large baking trays with
semolina. Divide the dough into four portions and roll each on a lightly
floured surface into a 20cm (8inch) round, about 5mm (¼inch) thick.
Place on the prepared trays. • Spread the tomato sauce over the bases,
then top with the artichokes, mozzarella and olives. Bake the pizzas for
18–20 minutes or until crisp and golden. Top with the basil leaves and
cut into wedges. • Drizzle the spinach, tomatoes and cucumber with the
vinegar and serve with the pizzas
Per serve
1790kJ/
426 calories; 21g protein; 9.5g fat (includes 3g saturated fat;
saturated : unsaturated fat ratio 0.46); 58g available carbs (includes
10g sugars and 48g starches); 11g fibre; 270mg sodium; 1150mg potassium;
sodium : potassium ratio 0.23
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Nutritional analysis To analyse Australian foods, beverages, processed products and recipes, we use FoodWorks which contains the AusNut and Nuttab databases. If necessary, this is supplemented with data from www.calorieking.com.au or http://ndb.nal.usda.gov/ndb/search.
Disclaimer GI News endeavours to check the veracity of news stories cited in this free e-newsletter by referring to the primary source, but cannot be held responsible for inaccuracies in the articles so published. GI News provides links to other World Wide Web sites as a convenience to users, but cannot be held responsible for the content or availability of these sites. All recipes that are included within GI News have been analysed however they have not been tested for their glycemic index properties by an accredited laboratory according to the ISO standards.
© ®™ The University of Sydney, Australia
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1 March 2018
GI News - March 2018
GI News is published by the University of Sydney,
School of Life and Environmental Sciences and the Charles Perkins
Centre. Our goal is to help people choose the high-quality carbs that
are digested at a rate that our bodies can comfortably accommodate and
to share the latest scientific findings on food and diet with a
particular focus on carbohydrates, dietary fibres, blood glucose and the
glycemic index.
Publisher: Professor Jennie Brand-Miller, AM, PhD, FAIFST, FNSA
Editor: Philippa Sandall
Scientific Editor/Managing Editor: Alan Barclay, PhD, APD AN
Contact GI News: glycemic.index@gmail.com
Sydney University Glycemic Index Research Service
Manager: Fiona Atkinson, PhD, APD AN
Contact: sugirs.manager@sydney.edu.au
Follow us on
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FOOD FOR THOUGHT
ANYTHING TO DECLARE?
Conflict of interest is about much more than money says Dr Arya Sharma. In a blog post in Obesity Notes
(a shortened version reprinted here with permission) he highlights the
conflicts and potential biases nutrition researchers should not hesitate
to acknowledge and disclose that John Ionnadis and John Trepanowski
raise in JAMA.
First, the authors focus on nutrition
research for good reasons. They say: “The totality of an individual’s
diet has important effects on health, [while] most nutrients and foods
individually have ambiguously tiny (or nonexistent) effects. Substantial
reliance on observational data for which causal inference is
notoriously difficult also limits the clarifying ability of nutrition
science. When the data are not clear, opinions and conflicts of interest
both financial and nonfinancial may influence research articles,
editorials, guidelines, and laws. Therefore, disclosure policies are an
important safeguard to help identify potential bias.”
While
the potential for financial conflict in relationship to the food
industry is well recognised and there are now well-established
disclosure norms, other conflicts, of which there are many, are not
routinely acknowledged, let alone, disclosed. For one, there are
significant financial conflicts that have nothing to do with taking
money from industry. For example: “Many nutrition scientists and experts
write books about their opinions and diet preferences. Given the
interest of the public in this topic, books about nutrition, diets, and
weight loss often appear on best-selling lists, even though most offer
little to no evidence to support their frequently bold claims.”
Furthermore: “Financial conflicts of interest can also
appear in unexpected places. For example, many not-for-profit nutrition
initiatives require considerable donor money to stay solvent. Public
visibility through the scientific literature and its reverberation
through press releases, other media coverage, and social media
magnification can be critical in this regard.”
But
conflicts can get even more complicated when they start reflecting
researchers’ own personal views and biases: “Allegiance bias and
preference for favorite theories are prevalent across science and can
affect any field of study. It is almost unavoidable that a scientist
eventually will form some opinion that goes beyond the data, and they
should. Scientists are likely to defend their work, their own
discoveries, and the theories that they proposed or espoused.” While
that is certainly true for any area of research, nutrition scientists
face an additional challenge.
“Every day they must make
numerous choices about what to eat while not allowing those choices to
affect their research. Most of them also have been exposed to various
dietary norms from their family, culture, or religion. These norms can
sometimes be intertwined with core values, absolutist metaphysical
beliefs, or both. For instance, could an author who is strongly adherent
to some religion conclude that a diet-related prescription of his or
her religion is so unhealthy as not to be worthwhile?”
The
authors propose that nutrition researchers: “disclose their advocacy or
activist work as well as their dietary preferences if any are relevant
to what is presented and discussed in their articles. This is even more
important for dietary preferences that are specific, circumscribed, and
adhered to strongly. For example, readers should know if an author is
strongly adherent to a vegan diet, the Atkins diet, a gluten-free diet, a
high animal protein diet, specific brands of supplements, and so forth
if these dietary choices are discussed in an article. The types of
articles in which relevant disclosure should be expected include
original research, reviews, and opinion pieces (such as editorials).”
Although the article focuses on nutrition research, the authors
acknowledge that similar biases may exist in other areas of research.
In
my own experience, ideological biases (although well-intended) are
pervasive through much of the research and publications on topics
ranging from physical activity to public health, where I often see
strong recommendations made based on evidence that is not even remotely
as robust or rigorous as the evidence that comes from, say a large
randomised clinical trials of a new prescription drug.
I
certainly agree with the authors’ recommendation that: “As a general
rule, if an author’s living example could be reasonably expected to
influence how some readers perceive an article, disclosure should be
encouraged. Authors who have strong beliefs and make highly committed
choices for diet or other behaviors should not hesitate to disclose
them. Doing so may help everyone understand who is promoting what and
why.”
Dr Arya M. Sharma, MD/PhD, FRCPC is Professor of Medicine & Chair in Obesity Research and Management at the University of Alberta, Edmonton, Canada. He is also the Clinical Co-Chair of the Alberta Health Services Obesity Program.
Read more:
• John Ionnadis and John Trepanowski: Disclosures in Nutrition Research: Why It Is Different.
• Dr Arya Sharma: Conflict Disclosures in Nutrition Research
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WHAT’S NEW?
MEDITERRANEAN DIET AND DEPRESSION
A
new trial from Deakin University shows improving diet quality helps
treat major depression. “We’ve known for some time that there is a clear
association between the quality of people’s diets and their risk for
depression,” says Professor Felice Jacka, president of the International
Society for Nutritional Psychiatry Research. “This is the case across
countries, cultures and age groups, with healthy diets associated with
reduced risk, and unhealthy diets associated with increased risk for
depression. However, this is the first randomised controlled trial to
directly test whether improving diet quality can actually treat clinical
depression.”
The researchers recruited adults with a
major depressive disorder and randomly assigned them to receive over a
three-month period either social support (helpful for people with
depression), or support from a clinical dietitian. The dietary group
were given information and help to improve their diet especially on
increasing vegetables, fruits, wholegrains, legumes, fish, lean red
meats, olive oil and nuts, and reducing sweets, refined cereals, fried
food, fast-food, processed meats and sugary drinks.
Participants
in the dietary intervention group had a much greater reduction in their
depressive symptoms over the three-month period, compared to those in
the social support group. At the end of the trial, a third of those in
the dietary support group met criteria for remission of major
depression, compared to 8 percent of those in the social support group.
“These results were not explained by changes in physical activity or
body weight, but were closely related to the extent of dietary change,”
says Jacka. “Those who adhered more closely to the dietary program
experienced the greatest benefit to their depression symptoms.
Importantly, depression also increases the risk of and, in turn, is also
increased by common physical illnesses such as obesity, type 2 diabetes
and heart disease. Successfully improving the quality of patients’
diets would also benefit these illnesses.”
Read more:
Pregnant women tend to be overwhelmed with unsolicited advice on a whole range of topics including “eat this, don’t eat that” dietary advice. Recently, a slew of fad diet books promoting low carb diets has cranked up the confusion about what to eat when you are expecting several notches by suggesting that by “reducing the intake of carbohydrates, [people] could significantly improve their health and well-being”. To set the record straight, a pregnant woman absolutely needs nutrient-rich sources of carbohydrate (what we call “good carbs”) in the right amounts to ensure she is meeting her own nutritional needs as well as her baby’s.
Prof Clare Collins and University of Newcastle researchers tackled six questions they are endlessly asked about eating carbs during pregnancy in a recent piece in The Conversation. They are now recruiting pregnant women in Australia (12–22 weeks gestation) to take part in an online survey to learn more about their nutrition knowledge and eating habits. The survey takes about 20–25 minutes and participants go into the draw to win one of four $100 gift cards. You can find out more about the survey and sign up to take part HERE.
Read more:
Six common questions about eating carbs during pregnancy answered
CHOOSING THE RIGHT DRINKS
Diabetes is a disease that develops when the body either stops producing the hormone insulin (type 1) or when the insulin it produces is not working properly (type 2). Insulin sensitivity describes how sensitive the body is to the effects of insulin, which is why it’s a key risk marker for diabetes. Someone who is insulin sensitive will require smaller amounts of insulin to lower blood glucose levels than someone who has low sensitivity to insulin.
A recent randomized controlled trial compared three everyday beverage choices on insulin sensitivity: semi-skimmed milk (recommended in dietary guidelines) with sugar-sweetened and “artificially” (intensely) sweetened soft drinks. Water was the non-calorie control. While the researchers hypothesized that drinking milk would improve insulin sensitivity and risk markers of cardiovascular disease in people who were overweight and obese, that’s not what they found. After 6 months’ intake of milk, or sugar-sweetened or intensely sweetened soft drinks, or water, there was no difference in risk markers for type 2 diabetes in the participants who remained weight stable throughout, suggesting the results were also independent of body weight.
Read more:
FEED THE BIRDS
In February, we wrote about how a dog can prompt you to be more active, help calm jagged nerves, and reduce feelings of isolation and loneliness. Interacting with birds is also good for us, mentally and physically, and people like to do this is by feeding the birds in their garden, “a habit that’s natural and extremely popular around the world,” says Prof Daryl Jones.
[Bird feeding] is a really important topic, says Jones, “because of the potential impact we’re having. We’re genuinely changing the shape of the wildlife community that lives in the city with us. These things we must not shy away from – if you are hosting people coming to your place to feed, you don’t allow them to go away sick. You are really, really, careful about how nutrient wise the food is and how clean the plate is. The same goes for birds.
As for a diet of bread and sugar or honey, traditional lorikeet fare in Australia, “that’s not a good idea at all,” says Jones, “as it can become very easily tainted with bacteria and fungi and make the birds ill. Probably the most important suggestion for bird feeders is to avoid mince and bread altogether; the mince is sticky, fatty and has too little calcium; far better is dry or tinned pet food. And bread is terrible for any animal apart from humans.”
Commenting on backyard bird feeding, Grainne Cleary, a researcher at Deakin University who led the Australian bird feeding and watering study, says “we need to understand its effect and provide guidelines to those who regularly feed birds in their backyards or urban settings.” She adds that making healthy food for birds more available on supermarket shelves, rather than just an ordinary cockatiel mix, will drastically improve bird health.
Darryl’s golden rules:
Cleanliness. Sweep up any left overs and spray feeder dish with a mix of water and vinegar. Dry and then add new feed. Daily.
Provide a snack. Not a meal. A little bit of healthy, appropriate food goes a long, long, way.
Never feed birds away from your home. Feeding bread to the ducks and swans at the park is a definite no-no.
Enjoy – because really, you're feeding the birds for yourself, not for their benefit.
Read more:
• Australian Bird Feeding and Watering Study
• Urban Bird Feeding: Connecting People with Nature
• To feed or not to feed
• Bird Feeding Takes Wing in U.S., With Summer Meals, Designer Seed
• Darryl Jones: The Birds at My Table. Why We Feed Wild Birds and Why It Matters (publishing March 2018)
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